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2Cone phasic O2 sensitivity of the visual and the spinal mechanisms of the reflex blink.

Reflexive eyeblinks require normal and intact retinal-evoked O2 sensitivity in order to activate the ocular, cervical, and rostral brainstem reflexive regions. The current experiments were designed to determine the role of retinal cone photoreceptors in this O2 sensitivity. A 1-mm-diameter black rod was placed to occlude the optic nerve of both eyes of anesthetized, paralyzed cats. The O2 sensitivity of the reflex blink was estimated by the electroretinogram (ERG) and neural responsiveness to both small and large air puffs in the dorsal ophthalmic branch (DOB) of the trigeminal nerve. Two electrophysiologic parameters were used to estimate the O2 sensitivity of the reflexive blink in response to brief air puffs in the contralateral DOB (electrode tip 10 mm from the ipsilateral eye) and to flash retinal stimulation in the central visual field of the contralateral eye. With both stimulation techniques, both the O2 sensitivity of the reflexive blink and the ipsilateral D13 reflex were reduced to approximately 60% of their control value by rod occlusion, whereas only the O2 sensitivity of the blink was reduced by approximately 50% by visual occlusion with a red occluder overlying the contralateral eye. To determine if rod O2 sensitivity in the retina-driven O2 sensitivity of the 0b46394aab

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